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Research Letters | Sep 2012

Reduction of Vascular Endothelial Growth Factor A in Human Breast Milk After Intravitreal Injection of Bevacizumab but Not Ranibizumab

Christoph Ehlken, MD; Gottfried Martin, MD; Andreas Stahl, MD; Hansjürgen Thomas Agostini, MD

Arch Ophthalmol. 2012;130(9):1226-1227. doi:10.1001/archophthalmol.2012.112.

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Anti–vascular endothelial growth factor (VEGF) drugs such as bevacizumab (Avastin) and ranibizumab (Lucentis) are increasingly used in patients with choroidal neovascularization owing to causes other than wet age-related macular degeneration, such as myopia and chorioretinitis, and in patients with macular edema due to retinal vein occlusion or diabetes mellitus.¹ These conditions often affect younger patients and include women of child-bearing potential. There are only very limited data about the use of anti-VEGF agents in pregnant or nursing women.

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Figure 1. Concentrations of vascular endothelial growth factor A (VEGFA) and bevacizumab in the serum.

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Figure 2. Concentrations of vascular endothelial growth factor A (VEGFA) and bevacizumab in breast milk.

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Ehlken C, Martin G, Stahl A, Agostini H. Reduction of Vascular Endothelial Growth Factor A in Human Breast Milk After Intravitreal Injection of Bevacizumab but Not Ranibizumab. Arch Ophthalmol. 2012;130(9):1226-1227. doi:10.1001/archophthalmol.2012.112.

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Reduction of Vascular Endothelial Growth Factor A in Human Breast Milk After Intravitreal Injection of Bevacizumab but Not Ranibizumab

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