Morning glory disc anomaly was first described by Kindler.1 Later, it was shown to be associated with retinal, cerebral, and carotid vascular anomalies.2 There are only a few large case series on morning glory disc anomaly and, to our knowledge, there is no previous documentation of peripheral nonperfusion.1- 5 Kindler first reported 10 patients with unusual congenital optic disc anomaly and named it “morning glory syndrome.” He noted white fluffy tissue in the optic disc center (100%), annulus retinal pigment epithelial disturbance around the optic disc (100%), multiple branching and radial vessels from the disc (70%), and retinal detachment (20%).1 Haik et al3 reported clinical features of 30 patients with morning glory disc anomaly, noting central glial tuft (100%) and associated retinal detachment (37%). Harasymowycz et al4 reported clinical features of 20 patients with morning glory disc anomaly and found disc telangiectasia (14%), vascular straightening and sheathing (14%), and retinal detachment (14%). Beyer et al,5 in 8 cases, noted vascular microtelangiectasia (67%), vascular sheathing (70%), vascular stretching (80%), and vascular looping (80%). On fluorescein angiography, arteriolar anastomoses near the optic disc were noted,5 but there was no comment on the peripheral retina. In these 4 case series and additional literature review by Lee and Traboulsi,6 there was no comment on peripheral fundus angiography and peripheral retinal nonperfusion.1- 5 Kim et al7 recently reported a case of unilateral peripapillary staphyloma that showed immature retina at birth in both eyes, but later, retinopathy of prematurity developed only in the eye with peripapillary staphyloma. They commented that retinal vascular development involves migration of astrocytes and a vascular precursor through the optic nerve; therefore, the optic nerve anomaly could affect the retinal vascular development.