Research Letters |

Traumatic Airbag Maculopathy

Jennifer Kung, MD; Loh-Shan B. Leung, MD; Theodore Leng, MD, MS; Y. Joyce Liao, MD, PhD
JAMA Ophthalmol. 2013;131(5):685-687. doi:10.1001/jamaophthalmol.2013.883.
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With the prevalence of motor vehicle crashes, airbag deployment is a significant source of ocular trauma. We describe a case of traumatic airbag maculopathy in which imaging studies document a constellation of interesting findings, including subretinal fluid with impending macular hole and persistent paracentral scotoma with underlying electrophysiological disturbance despite anatomical recovery on optical coherence tomography (OCT).

Correspondence: Dr Leng, Byers Eye Institute at Stanford, 2452 Watson Ct, Palo Alto, CA 94303 (tedleng@stanford.edu).

Conflict of Interest Disclosures: None reported.

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Figure 1. At presentation, several areas of retinal opacification around the disc on a fundus photograph (A) corresponded to foci of decreased autofluorescence on fundus autofluorescence (B). Notably, fundus autofluorescence also shows a diffuse area of increased autofluorescence around the fovea extending superiorly toward the nerve. This distribution of photoreceptor injury appears as a near total loss of electroretinographic signal in the same pattern a year later (C).

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Figure 2. Optical coherence tomography suggested an impending macular hole at presentation (A). The retinal architecture recovered by 3 weeks (B), and at 5 months it continued to show preservation of the external limiting membrane, inner segment–outer segment junction, and foveal cone structure (C).




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