The number of legally blind eyes did not differ between patients with necrotizing and those with nonnecrotizing variants of herpetic uveitis (15 of 29 [52%] and 2 of 9 [22%], respectively; P = .15), nor between patients from the ARN and non-ARN groups (13 of 25 [52%] and 4 of 13 [31%], respectively; P = .31; Table 3). The most important reasons for loss of vision in the ARN group were RD (7 of 13 [54%]) and retinal atrophy with scarring (6 of 13 [46%]). The reasons for vision loss in the non-ARN group were retinal atrophy (2 of 4 [50%]), RD (1 of 4 [25%]), and undetermined (1 of 4 [25%]). In all 38 patients with herpetic uveitis, VA of less than 0.1 was associated with the presence of occlusive vasculitis (P = .04) and RD (P = .01); the latter 2 variables were not associated with each other. The presence of occlusive vasculitis was associated with worse VA at 6 months' follow-up; patients with vasculitis had, on average, a VA of 0.05 compared with 0.5 in patients without vasculitis (P = .001). In our series, VA of less than 0.1 was not associated with sex, age, viral species, hypotony, cystoid macular edema, presence of necrotic lesions or speed of progression of these lesions, or time to diagnosis. In patients in the ARN group, the start of antiviral treatment longer than 3 weeks after the onset of symptoms was associated with worse VA at 6 months' follow-up (VA of 0.05 and 0.3; P = .03). In patients in the non-ARN group, the VA at 6 months was 0.8 when treated within 3 weeks of the onset of symptoms and 0.3 when treated after 3 weeks of onset (P = .11).